这篇经验文章会给广大网友说明一下“喷施草甘膦后可以播种吗”的内容进行周详介绍，希望对各位农友们稍微有点帮助，赶紧收藏吧！
1、草甘膦使用后多久可以播种？1、使用草甘膦后需要等待一定时间才能进行播种。2、草甘膦是一种广谱性的除草剂，可以控制许多杂草和草本植物，但对于部分作物也会产生影响。草甘膦使用后需要等待一定时间，让它在土壤中分解，避免对作物造成伤害。3、草甘膦使用后需要根据不同的作物和使用情况来确定具体的等待时间。通常情况下，需要等待至少一周以上，甚至可能需要等待数周至数月不等才能进行播种。这个时间也会受到气温、降雨等环境因素的影响。在使用草甘膦前，需要认真了解相关的使用说明，并按照要求来做好田间管理工作。
2、草甘膦打过的菜地要几天才可以种菜？我的意见是至少要间隔10-15天之后种菜才会有比较好的安全性，对蔬菜苗和种子的影响才能最小、安全性能相对的好一些。
草甘膦的一个特点就是遇土钝化、分解。但是分解之后的产物是否会对农作物的根系或者种子产生影响，这个问题现在还没有明确的解释吧。而且，现在很多种植果树使用草甘膦灭草的果农也都在反馈果园连续使用草甘膦2-3年会出现挂果过早黄化的情况。所以，草甘膦即便是遇土分解或者钝化了，其安全性还是有待商榷。
特别是蔬菜作物，相对的来说对除草剂较为敏感，极易出现除草剂药害的情况。所以，如果喷施草甘膦之后间隔时间过短就移栽或者播种蔬菜的种子，蔬菜苗的根系或者幼芽受草甘膦药害的风险也是比较大的。但是间隔的时间越长，经过土壤的理化作用以及土壤微生物的作用之后，应该可以减少土壤中草甘膦分解后的残留物的基数，安全性方面也会更好。
以上是我对这个问题的一些看法，如有不当之处请海涵。
3、喷草甘膦后再种树苗有害？
喷了草甘膦以后，可以种植树苗，最好是喷草甘膦以后10天再种树苗，那样的话就基本没有甚么药害了。
一般来说15天之后吧，但是一些果树还是多隔一段时间。望采纳它是内吸性灭生除草剂，可广泛用于非耕地，山地林地，深根性果园等灭生性除草。屠欢具有杀草谱广、低温效果稳定、除草彻底、土壤残留小等优良特性，是一款高效、安全、彻底、持久的泛用性灭生除草剂。但是1.葡萄、猕猴桃、杨梅、香蕉、火龙果、桑园等浅根系果园禁用；
2.瓜类、叶菜类、玉米等浅根系作物行间禁用；
3.后茬种植（含直播、移栽）禾谷类作物（水稻、小麦）以及棉花、大豆、花生，间隔7天；
4.后茬种植（含直播、移栽）葱、姜、蒜、十字花科蔬菜，间隔15天；
5.玉米用药后1天可以播种，播后苗前情况下用药安全。
Previous stu**s of Escherichia coli 5-enolpyruvoylshikimate-3-phosphate synthase (EPSPS, EC 2.5.1.19) have suggest** that the kinetic reaction mechani** for this enzyme in the forward direction is equilibrium order** with shikimate 3-phosphate (S**) binding first follow** by phosphoenolpyruvate (PEP). Recent results from this laboratory, however, measuring direct binding of PEP and PEP **ogues to free EPSPS suggest more random character to the enzyme. Steady-state kinetic and spectroscopic stu**s present** here indicate that E. coli EPSPS does inde** follow a random kinetic mechani**. Initial velocity stu**s with S** and PEP show competitive substrate inhibition by PEP add** to a normal intersecting pattern. Substrate inhibition is propos** to occur by competitive binding of PEP at the S** site [Ki(PEP) = 6-8 mM]. To test for a productive EPSPS.PEP binary complex, the reaction order of EPSPS was evaluat** with shikimic acid and PEP as substrates. The mechani** for this reaction is equilibrium order** with PEP binding first giving a Kia value for PEP in agreement with the independently measur** Kd of 0.39 mM (shikimate Km = 25 mM). Results from this study also show that the 3-phosphate moiety of S** offers 8.7 kcal/mol in binding energy versus a hydroxyl in this position. Over 60% of this binding energy is express** in binding of substrate to enzyme rather than toward increasing kcat. Glyphosate inhibition of shikimate turnover was poor with approximately 8 x 10(4) loss in binding capacity compar** to the normal reaction, consistent with the independently measur** Kd of 12 mM for the EPSPS.glyphosate binary complex. The EPSPS.glyphosate complex induces shikimate binding, however, by a factor of 7 greater than EPSPS.PEP. Carboxyallenyl phosphate and (Z)-3-fluoro-PEP were found to be strong inhibitors of the enzyme that have surprising affinity for the S** binding domain in addition to the PEP site as measur** both kinetically and by direct observation with 31P NMR. The collective data indicate that the true kinetic mechani** for EPSPS in the forward direction is random with synergistic binding occurring between substrates and inhibitors. The synergi** explains how the mechani** can be random with S** and PEP, but yet equilibrium order** with PEP binding first for shikimate turnover. Synergi** also accounts for how glyphosate can be a strong inhibitor of the normal reaction, but poor versus shikimate turnover.